Pathogenic chaperone-like RNA induces congophilic aggregates and facilitates neurodegeneration in Drosophila

  • Elena Savvateeva-Popova
  • , Andrei Popov
  • , Abraham Grossman
  • , Ekaterina Nikitina
  • , Anna Medvedeva
  • , Alexander Peresleni
  • , Leonid Korochkin
  • , James G. Moe
  • , Eliot Davidowitz
  • , Konstantin Pyatkov
  • , Elena Myasnyankina
  • , Olga Zatsepina
  • , Natalia Schostak
  • , Elena Zelentsova
  • , Michael Evgen'ev

Research output: Contribution to journalArticlepeer-review

Abstract

Protein aggregation is a hallmark of many neurodegenerative diseases. RNA chaperones have been suggested to play a role in protein misfolding and aggregation. Noncoding, highly structured RNA recently has been demonstrated to facilitate transformation of recombinant and cellular prion protein into proteinase K-resistant, congophilic, insoluble aggregates and to generate cytotoxic oligomers in vitro. Transgenic Drosophila melanogaster strains were developed to express highly structured RNA under control of a heat shock promoter. Expression of a specific construct strongly perturbed fly behavior, caused significant decline in learning and memory retention of adult males, and was coincident with the formation of intracellular congophilic aggregates in the brain and other tissues of adult and larval stages. Additionally, neuronal cell pathology of adult flies was similar to that observed in human Parkinson's and Alzheimer's disease. This novel model demonstrates that expression of a specific highly structured RNA alone is sufficient to trigger neurodegeneration, possibly through chaperone-like facilitation of protein misfolding and aggregation.

Original languageEnglish
Pages (from-to)9-19
Number of pages11
JournalCell Stress and Chaperones
Volume12
Issue number1
DOIs
StatePublished - Mar 2007
Externally publishedYes

ASJC Scopus subject areas

  • Biochemistry
  • Cell Biology

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