Mitochondrial Superoxide Contributes to Hippocampal Synaptic Dysfunction and Memory Deficits in Angelman Syndrome Model Mice

Keyphrases

• Angelman Syndrome 100%
• Memory Deficits 100%
• Syndrome Model 100%
• Synaptic Dysfunction 100%
• Synaptic Memory 100%
• Mitochondrial Superoxide 100%
• UBE3A Gene 37%
• MitoQ 37%
• Neurodevelopmental Disorders 25%
• Oxidative Stress 25%
• Mitochondrial Reactive Oxygen Species (mtROS) 25%
• Hippocampus 12%
• Pathophysiology 12%
• Hippocampal Synaptic Plasticity 12%
• Synaptic Plasticity 12%
• Speech Motor 12%
• Mitochondria 12%
• Contextual Fear Memory 12%
• Neurodegenerative Diseases 12%
• Epilepsy 12%
• Ubiquitin Ligase 12%
• E6AP 12%
• Antioxidant 12%
• Developmental Delay 12%
• Motor Dysfunction 12%
• Area CA1 12%
• Chromosome 15 12%
• 26S Proteasome 12%
• Superoxide 12%
• Paternal Allele 12%
• Mitochondrion-specific 12%

Neuroscience

• Angelman Syndrome 100%
• Amnesia 100%
• Synaptic Plasticity 25%
• Mitochondrion 25%
• Oxidative Stress 25%
• Reactive Oxygen Species 25%
• Hippocampus 12%
• Stereotypic Movement Disorder 12%
• Neurodevelopmental Disorder 12%
• Ubiquitin Protein Ligase 12%
• Neurodegenerative Disorder 12%
• Antioxidant 12%
• Chromosome 15 12%
• Proteasome 12%

Biochemistry, Genetics and Molecular Biology

• Mouse Model 100%
• Angelman Syndrome 100%
• UBE3A 37%
• Nerve Cell Plasticity 25%
• Oxidative Stress 25%
• Mitochondrion 25%
• Allele 12%
• Enzyme 12%
• Proteasome 12%
• Ubiquitin Ligase 12%
• Mitochondrial-Targeting 12%
• Chromosome 15 12%