Curcumin ameliorates acute thioacetamide-induced hepatotoxicity

Keyphrases

• Antioxidant 10%
• Biochemical Parameters 10%
• Blood Ammonia 10%
• Curcumin 100%
• Fulminant Hepatic Failure 30%
• Hepatic Necroinflammation 20%
• Hepatocellular Injury 10%
• High Dose 20%
• Improved Survival 10%
• Induced Hepatotoxicity 100%
• Inducible Nitric Oxide 10%
• Inducible Nitric Oxide Synthase 30%
• Insults 10%
• Intraperitoneal Injection 10%
• Liver 10%
• Liver Disease 10%
• Liver Injury 10%
• Nitric Oxide 10%
• Nitric Oxide Production 10%
• Nuclear Binding 10%
• Nuclear factor-B 50%
• Oxidative Stress 20%
• Protectins 10%
• Protein Expression 10%
• Rat Model 20%
• Reactive Oxygen Species Production 10%
• Survival Rate 10%
• Therapeutic Potential 10%
• Thioacetamide 100%
• Thioacetamide Hepatotoxicity 10%
• Thiobarbituric Acid Reactive Substances 10%

Pharmacology, Toxicology and Pharmaceutical Science

• Alternative Complement Pathway C3 C5 Convertase 50%
• Antioxidant 10%
• Curcumin 100%
• Fulminant Hepatic Failure 30%
• Hepatotoxicity 100%
• Intraperitoneal Injection 10%
• Liver Cell Damage 10%
• Liver Disease 10%
• Liver Injury 10%
• Nitric Oxide 30%
• Nuclear Factor 50%
• Rat Model 20%
• Reactive Oxygen Metabolite 10%
• Survival Rate 10%
• TBARS 10%
• Thioacetamide 100%

Immunology and Microbiology

• Ammonia Blood Level 20%
• Complement Factor B 100%
• Drug Megadose 40%
• Low Drug Dose 40%
• Oxidative Stress 40%
• Protein Expression 20%
• Rat Model 40%
• Survival Rate 20%