Cannabinoids prevent the development of behavioral and endocrine alterations in a rat model of intense stress

Keyphrases

• Basolateral Amygdala 100%
• Rat Model 100%
• WIN55,212-2 100%
• Cannabinoids 100%
• Acoustic Startle Reflex 100%
• Behavior Modification 100%
• Single Prolonged Stress 100%
• Endocrine Alterations 100%
• Inhibitory Avoidance 75%
• Posttraumatic Stress Disorder 50%
• Stress Disorders 50%
• Posttraumatic Stress 50%
• CB1 Receptor 50%
• Avoidance Conditioning 50%
• HPA Axis 50%
• Avoidance Extinction 50%
• Stressful Events 25%
• Stress-induced 25%
• Anxiety Disorders 25%
• Level of Functioning 25%
• Anxiety Level 25%
• Preventive Effect 25%
• Potentiation 25%
• Receptor Activation 25%
• CB1 Receptor Antagonist 25%
• AM251 25%
• Cannabinoid Receptor 1 (CB1R) 25%
• Pharmacological Treatment 25%
• Stress Exposure 25%
• Negative Feedback 25%
• Intervention Therapy 25%
• Potential Treatments 25%
• Point Following 25%
• CB2 Receptor Agonist 25%
• Conditioned Avoidance Response 25%
• Hypothalamic-pituitary-adrenal Axis Function 25%
• Single Prolonged Stress Model 25%
• Optimal Window Size 25%
• Traumatic Stress 25%

Neuroscience

• Basolateral Amygdala 100%
• Startle Response 100%
• Cannabinoid 100%
• Posttraumatic Stress Disorder 75%
• Hypothalamic-Pituitary-Adrenal Axis 75%
• Cannabinoid 1 Receptor 50%
• Avoidance Behavior 50%
• Avoidance Response 50%
• CB1 Receptor Antagonists 25%
• AM251 25%
• Negative Feedback 25%
• Cannabinoid Receptor 25%
• CB2 Receptor Agonist 25%
• Drug Therapy 25%