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ACKR2 limits skin fibrosis and hair loss through IFN-β
Sergei Butenko
, Nofar Ben Jashar
, Tsofiya Sheffer
, Edmond Sabo
, Sagie Schif-Zuck
,
Amiram Ariel
Department of Biology
Department of Human Biology
Research output
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Contribution to journal
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Article
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peer-review
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Keyphrases
Hair Loss
100%
ACKR2
100%
Skin Fibrosis
100%
Inflammation Resolution
33%
Pro-resolving
33%
Inflammatory Chemokines
33%
Profibrotic
33%
Hair Follicle
16%
Epidermis
16%
Interleukin-12 (IL-12)
16%
Interleukin-10
16%
12/15-Lipoxygenase
16%
Tissue Repair
16%
Skin Lesions
16%
Wound Healing
16%
Atrophy
16%
Bleomycin
16%
Effector Molecules
16%
Pro-resolving Mediators
16%
Collagen Deposition
16%
Subcutaneous Adipose Tissue
16%
WT Mice
16%
Disorientation
16%
Lesion Size
16%
Immune Stimulation
16%
IFN Production
16%
Chemokine Receptor 2
16%
Acute Inflammatory
16%
Skin Section
16%
Profibrotic Cytokines
16%
Inflammatory Skin
16%
Atypical Chemokine Receptor
16%
Fibrotic Scar
16%
Chemokine CCL2
16%
Pharmacology, Toxicology and Pharmaceutical Science
Cytokine
100%
Skin Fibrosis
100%
Hair Loss
100%
Inflammation
66%
Scar Formation
33%
Tissue Repair
33%
Arachidonate 15 Lipoxygenase
33%
Wound Healing
33%
Chemokine
33%
Bleomycin
33%
Wild Type Mouse
33%
Interleukin 10
33%
Chemokine Receptor
33%
Skin Defect
33%
Monocyte Chemotactic Protein 1
33%
Disorientation
33%
Medicine and Dentistry
Cytokine
100%
Skin Fibrosis
100%
Mediator
66%
Hair Follicle
33%
Adipose Tissue
33%
Arachidonate 15 Lipoxygenase
33%
Tissue Repair
33%
Scar Formation
33%
Skin Defect
33%
Bleomycin
33%
Wound Healing
33%
Disorientation
33%
Chemokine
33%
Chemokine Receptor
33%
Monocyte Chemotactic Protein 1
33%
Interleukin 10
33%
Immunology and Microbiology
Cytokine
100%
Mediator
66%
Chemokine
33%
Interleukin 10
33%
Tissue Repair
33%
Scar Formation
33%
Wound Healing
33%
Adipose Tissue
33%
Chemokine Receptor
33%
Wild Type Mouse
33%
Hair Follicle
33%
Monocyte Chemotactic Protein 1
33%
Lipoxygenase
33%